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New York Times

The British variant of coronavirus: what we know

In recent days, the world has observed with curiosity and scars alarms the scientists of the United Kingdom describes a recent variant identified by coronavirus that appears to be more contagious than other known, in addition to genetically distinct. Initial studies of the new variant will take place as Prime Minister Boris Johnson reiterates the restrictions on Navigation and exhorts the authorities of the Netherlands, Germany and other European nations to ban travel from the United Kingdom. At the moment, the new variant is the focus of intense debates and analyzes. This is something that scientists have found just now. Is the United Kingdom variant a new supervirus species? No. Only a variety of strains have been treated that have been treated in accordance with the coronavirus SARS-CoV-2 have been propagated worldwide. The mutations present when the virus replicates, and this variant — known as B.1.1.7 — has its own characteristic conjunctive of them. What is unusual? The variant called for the attention of investigators in December, when it began appearing with the highest frequency in two countries in the regions of southern England. As a result, many patients have been living since September. When investigators observed searching their genome, they were affected by the relatively large amount of mutations that developed: 23 i alt. The majority of mutations that surge in the coronavirus are due to the virus having no effect. Without embarrassment, I get the impression that variations of the mutations in the B.1.1.7 can affect the form of virus propagation. Is it more contagious than other viruses? This is a parece. In preliminary studies, investigators from the United Kingdom found that the virus was spreading rapidly in parts of the English and that there was a large number of other variants that had been circulating for months. Without embarrassment, the heck out of which a bit of the virus is spreading every now and then is not proven to be propelled by more speed than others. Can only be given solely for questions about azar. For example, the powder can surpass a variant within a very thick paper, which can be transmitted with ease and, for that matter, create more copies of its own. However, epidemiologic evidence has now recovered from what the English believe is that this variant is very good for propagation. In some places where it is most common, there is a total amount of coronavirus cases. Neil Ferguson, an investigator specializing in public health at the Imperial College of London, considers that the high transmission rate of this variant is between 50 and 70 per cent, and compares with other variants in the United Kingdom. Some scientists have considered the possibility that the increase in transmission is the result, at least in part, of infecting the nines. Normally, children are less likely to fight and transmit the virus than adolescents or adults. Without embarrassment, the new chapter could turn into “suspicious as adults”, commented by Wendy Barclay, governor of the governing body and virologist of the Imperial College de Londres. Provoke a more serious illness? Nothing is evident from the evidence that it confirms, at least now. No hindrance, hay razones para tomar en serio la posibilidad. In South Africa, another coronavirus virus has developed a mutation in particular that also occurs in B.1.1.7. This variant is propagated to all velocities across the coastal zones of South Africa. In addition, in preliminary studies, doctors will find that the vaccine infected with this variant carries an intensified viral load: a higher concentration of the virus in its superior respiratory tract. In many viral diseases, it is associated with more serious symptoms. ¿De dónde viene esta variante ususual? Hø a debate intenso and torno a esa pregunta. One possibility is that the variant has been unveiled in its series of new mutations in the interior of a special set of pedigrees. In a typical infection, the people contract the coronavirus and become contagious during many days before manifesting symptoms. Luego, the virus itself becomes abundant in the body, in accordance with the humanitarian system commanding a defense. Although patients suffer from a serious case COVID-19, suelen librarse del virus fully in a few weeks as much. No hindrance, in veces el-virus infects a gente con systems of immune systems débiles. In its entirety, the virus can develop during months. Case studies on these immunocompromised individuals have shown that the virus can accumulate a large amount of mutations that replicate in their bodies over a long period of time. Some investigators have found that, over time, natural selection could favor the mutant viruses that evoke the humanitarian system. He also suggested that the evolution of the variant variant should have received an additional impulse from the medicine that these patients take. It is possible that some mutants are tolerant of drugs such as monoclonal antibodies. Other scientists have reported that the virus itself has developed new mutations to propagate through medium-sized animal populations, such as visions, before they could enter the human population. These “animal reservoirs” have a vested interest in conforming with their detection of animal infections. Is the variant circulating in the United States? Todavía nej, hasta donde se sabe. Syndembargo, it is not clear whether it has been linked to the United States. British scientists have set up a much better system to monitor coronavirus for new mutations. It can be said that it has been transferred from the United Kingdom to the transaction. Now that the world knows that it must look for the variant, it will be possible to appear in more lands. Will the variant become ineffective in the new vacancies? No. The majority of experts doubt that they have any significant effect on vacancies, although it is not possible to say that they can have any effect. The Food and Drug Administration has authorized vacancies, one of Modern and the other of Pfizer and BioNTech. Ambass vacunas skaber immunoadad en contra del coronavirus al ensñarles a nuestros immunitarios a crear anticuerpos para une protein, llamada de spiga, que se encuentra en la surfaceie vir virus. The spit protein accumulates in the cells and opens a passageway through the interior. The antique products produced in response to the vacancies are at the point of delivery. The result: the virus can not enter. It can be a mutation in a coronavirus that changes the shape of its sphincter proteins, which dictates that the antibodies are well related to them. In addition, the mutations in B.1.1.7 are also encapsulated in the gene of spike. However, our human immune systems can produce an entire range of anticoagulants in contrast to a single viral protein, which is likely to cause viruses to escape the ability to attack. At the moment, experts do not believe that the variant can evade vacancies. To confirm this, investigators from the Walter Reed Institute of Investigation are analyzing the changes in the structure of their protein. This article originally appeared in The New York Times. © 2020 The New York Times Company